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Abnormal Prefrontal Neural Oscillations are Associated with Social Deficits in MECP2 Duplication Syndrome
文献摘要:
Dear Editor, Methyl-CpG-binding protein 2(MeCP2)participates in regulating the expression of the CREB,TDP-43 and Dnmt1 genes[1,2].Recent studies have shown that MeCP2 is essential for maintaining normal neural functions in the mammalian brain.Overexpression of MeCP2 causes MECP2 duplication syndrome[3],a type of autism spectrum disorder that results in a core phenotype of social deficits in human patients[4],nonhuman primates[5],and rodents[6].
文献关键词:
作者姓名:
Xiao Li;Yingnan Nie;Qiyu Niu;Xuanjun Guo;Zilong Qiu;Shouyan Wang
作者机构:
Institute of Intelligent Robotics,Academy for Engineering and Technology,Fudan University,Shanghai 200433,China;Institute of Science and Technology for Brain-Inspired Intelligence,Fudan University,Shanghai 200433,China;Center for Excellence in Brain Science and Intelligence Technology,Institute of Neuroscience,State Key Laboratory of Neuroscience,CAS Key Laboratory of Primate Neurobiology,Chinese Academy of Sciences,Shanghai 200031,China;National Clinical Research Center for Aging and Medicine,Huashan Hospital,Fudan University,Shanghai 200040,China
引用格式:
[1]Xiao Li;Yingnan Nie;Qiyu Niu;Xuanjun Guo;Zilong Qiu;Shouyan Wang-.Abnormal Prefrontal Neural Oscillations are Associated with Social Deficits in MECP2 Duplication Syndrome)[J].神经科学通报(英文版),2022(12):1598-1602
A类:
Deficits, Methyl
B类:
Abnormal,Prefrontal,Neural,Oscillations,are,Associated,Social,MECP2,Duplication,Syndrome,Dear,Editor,CpG,binding,protein,MeCP2,participates,regulating,CREB,TDP,Dnmt1,genes,Recent,studies,have,shown,that,essential,maintaining,neural,functions,mammalian,brain,Overexpression,causes,duplication,syndrome,autism,spectrum,disorder,results,core,phenotype,social,deficits,patients,nonhuman,primates,rodents
AB值:
0.733243
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